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- When the body gets low on water, the hypothalamus increases the synthesis of an antidiuretic hormone called vasopressin, which is secreted by the pituitary gland and travels to the kidneys. There, it causes water to be reabsorbed from the urine, thus reducing urine flow and conserving water in the body until more fluids are consumed.
www.brainfacts.org/Archives/2008/The-Neural-Regulation-of-Thirst
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Sep 26, 2019 · Figure 1: Brain regions controlling thirst. The lamina terminalis (yellow) is a series of interconnected brain structures that act as a central hub to control fluid levels in the body. Some cells in the lamina terminalis are adjacent to large, fluid-filled compartments in the brain, called ventricles (blue).
Mar 16, 2008 · Scientists have been studying the neurological mechanisms of thirst for decades. Early on, they discovered that the body’s primary “thirst center” in the brain is the hypothalamus, a deep structure that also regulates body temperature, sleep, and appetite.
The key brain structure for the genesis of thirst is the lamina terminalis (LT), a group of three deep forebrain nuclei that coordinate the homeostatic response to fluid imbalance (described in more detail below).
- Christopher A. Zimmerman, David E. Leib, Zachary A. Knight
- 10.1038/nrn.2017.71
- 2017
- 2017/08
Jan 1, 2020 · A brain structure called lamina terminalis (LT) in the mammalian forebrain is the primary site that regulates thirst. In the LT, neurons that control water intake are housed in three anatomically linked nuclei—the subfornical organ (SFO), the organum vasculosum of LT, and the median preoptic nucleus (MnPO).
- Vineet Augustine, Vineet Augustine, Sangjun Lee, Yuki Oka
- 10.1016/j.cell.2019.11.040
- 2020
- 2020/01/01
Jun 22, 2017 · Thirst is a homeostatic response to changes in fluid balance and is governed by a set of interconnected brain structures known as the lamina terminalis.
- Christopher A. Zimmerman, David E. Leib, Zachary A. Knight
- 2017
Dec 19, 2016 · In this Primer, Leib et al. discuss the physiological basis of thirst, including the mechanisms by which the brain monitors the state of the blood, how dedicated neural circuits control drinking behavior in diverse contexts, and the interplay between thirst and other homeostatic processes, like hunger.
They proposed three mechanisms: first, that neuron activity decreases due to the anticipation of water; second, that neuron activity decreases as water is consumed; and third, that neuron activity continues until a certain satiety threshold is reached, after which it abruptly turns off.
